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specific pathways are activated or inhibited by specific SCBs. Due to the wide range of chemical structures discovered as cannabinoid agonists, it is very plausible that structurally distinct ligands bind to CB1Rs in a unique methods. Receptor conformations resulting with the binding of individual ligands, or several, at the same time binding ligands, might dictate the kind, strength and variety of G-proteins activated because of the receptor. The variability of possible cannabinoid receptor-ligand complexes, along with the numerous downstream pathways That could be differentially activated or inhibited, provide ample chance for functional selectivity, which certainly is properly proven in cannabinoid receptor signaling (Bosier et al.
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